Antibodies Antineutrophil Cytoplasmic
Mostrando 1-12 de 48 artigos, teses e dissertações.
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1. Lupus-like nephritis with positive anti-neutrophil cytoplasmic antibodies and negative antinuclear antibodies
Resumo Os anticorpos anticitoplasma de neutrófilos (ANCAs) estão associados à vasculite de pequenos vasos, no entanto, a sua prevalência não é rara em outras doenças imunológicas. Na nefrite lúpica (LN), o seu papel patológico e relevância clínica têm sido alvo de pontos de vista controversos. Apresentamos um caso de lesão renal aguda e síndro
Brazilian Journal of Nephrology. Publicado em: 2022
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2. Antineutrophil cytoplasmic antibodies in patients treated with methimazole: a prospective Brazilian study
Resumo Introdução: Os efeitos adversos de drogas antitireoidianas são conhecidos. Vasculite associada a anticorpos anticitoplasma de neutrófilos é uma reação adversa grave. A maioria dos estudos que avaliam anticorpos anticitoplasma de neutrófilos relacionado a drogas antitireoidianas envolveu pacientes tratados com propiltiouracil, entretanto menos
Braz. j. otorhinolaryngol.. Publicado em: 07/11/2019
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3. Anca negative pauci-immune crescentic glomerulonephritis and mixed connective tissue disease: a case study
Resumo Uma das causas mais comuns da glomerulonefrite rapidamente progressiva (GNRP) é a glomerulonefrite crescêntica (GNC) pauci-imune. Na maioria dos casos, a patologia apresenta um marcador sorológico positivo, o anticorpo anticitoplasma de neutrófilos (ANCA), mas em cerca de 10% dos pacientes não há ANCAs circulantes, perfazendo um subgrupo da pato
J. Bras. Nefrol.. Publicado em: 18/03/2019
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4. Anti-neutrophil cytoplasmic antibodies in new-onset systemic lupus erythematosus
Abstract: Background: The clinical significance of anti-neutrophil cytoplasmic antibodies in patients with new-onset systemic lupus erythematosus, especially in systemic disease accompanied by interstitial lung disease remains to be elucidated. Objectives: This study was designed to investigate the role of anti-neutrophil cytoplasmic antibodies in new-onse
An. Bras. Dermatol.. Publicado em: 2017-08
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5. Henoch-Schönlein purpura with c-ANCA antibody in an adult
Abstract: The Henoch-Schönlein purpura is the vasculitis associated with deposits of immunoglobulin A in small vessels. Its association with cytoplasmic antineutrophil cytoplasmic antibodies is possible, but rare. This vasculitis is uncommon in adults and the main clinic manifestations are purpuric lesions in lower limbs with gastrointestinal symptoms and r
An. Bras. Dermatol.. Publicado em: 2016-10
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6. Cocaine/levamisole-induced systemic vasculitis with retiform purpura and pauci-immune glomerulonephritis
Levamisole has been increasingly used as an adulterant of cocaine in recent years, emerging as a public health challenge worldwide. Levamisole-associated toxicity manifests clinically as a systemic vasculitis, consisting of cutaneous, hematological, and renal lesions, among others. Purpura retiform, cutaneous necrosis, intravascular thrombosis, neutropenia,
Braz J Med Biol Res. Publicado em: 26/04/2016
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7. Granulomatosis with polyangiitis, a new nomenclature for Wegener's Granulomatosis - Case report
AbstractThe granulomatosis with polyangiitis, initially known as Wegener's granulomatosis, is a small and medium vessels vasculitis. It's classic form presents a triad: necrotizing granuloma of respiratory tract, necrotizing cutaneous vasculitis and glomerulonephritis. This vasculitis has cytoplasmic antineutrophil antibodies as signal. This work illustrates
An. Bras. Dermatol.. Publicado em: 2015-06
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8. Study of microvascular thrombosis in lung biopsies of patients with Wegeners granulomatosis / Estudo da trombose microvascular em biópsias pulmonares de pacientes com granulomatose de Wegener
Wegeners granulomatosis (GW) is associated with thromboembolic events. In this work, we quantified the thrombus in small/medium-sized pulmonary arteries of patients with GW (n:24) compared to normal controls (n:16). The results showed that the GW and control arteries were similar regarding total area. The thrombus area was significantly increased in GW compa
Publicado em: 2008
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9. Evaluation of Antineutrophil cytoplasmic autoantibodies (ANCA) and Anti-Saccharomyces cerevisiae antibodies (ASCA) in inflammatory bowel disease / Pesquisa de anticorpos contra estruturas citoplasmáticas do neutrófilo (ANCA) e contra o Saccharomyces cerevisiae (ASCA) na doença inflamatória intestinal
A determinação dos marcadores sorológicos P-ANCA (anticorpo perinuclear contra estruturas citoplasmáticas do neutrófilo) e ASCA (anticorpo anti-Saccharomyces cerevisiae) auxilia de forma menos invasiva no diagnóstico da doença inflamatória intestinal (DII). O padrão de associação mais relacionado à retocolite ulceratica inespecífica (RCUI) ocorr
Publicado em: 2007
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10. Detection of anti-neutrophil cytoplasmic antibodies after acute Plasmodium falciparum malaria.
Four of 30 patients with Plasmodium falciparum infection in Bangkok, Thailand, were positive for anti-neutrophil cytoplasmic antibodies by indirect immunofluorescence 1 month after antimalarial therapy. No myeloperoxidase, proteinase 3, lactoferrin, or elastase reactivity was found. Since no evidence of vasculitis was seen in these patients, anti-neutrophil
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11. Formalin fixation and patterns of antineutrophil cytoplasmic antibodies.
Recent publications have suggested that fixing neutrophils in formalin is a useful adjunct to the differentiation of antinuclear antibodies from perinuclear antineutrophil cytoplasmic antibodies. In this small comparative study of 30 sera the use of an additional slide of formalin fixed neutrophils did not add any useful information and may be confusing as t
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12. Idiopathic pulmonary haemosiderosis: a form of microscopic polyarteritis?
Idiopathic pulmonary haemosiderosis remains a diagnosis of exclusion in patients who present with pulmonary alveolar haemorrhage. Systemic vasculitis developed in a patient with an eight year history of idiopathic pulmonary haemosiderosis. The diagnosis was confirmed by a rising titre of antineutrophil cytoplasmic antibodies directed against myeloperoxidase.