Evidence that glucose starvation-sensitive mutants are altered in the relB locus.
AUTOR(ES)
Mosteller, R D
RESUMO
Genetic mapping studies indicate that the relaxed-control mutants isolated on the basis of sensitivity to glucose starvation contain lesions in the relB locus. These mutants, which are sensitive to protein synthesis inhibitors such as sulfacetamide, exhibit relaxed control of both RNA and phospholipid syntheses.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=222118Documentos Relacionados
- Genetics of the relB locus in Escherichia coli.
- Purification of the RelB and RelE Proteins of Escherichia coli: RelE Binds to RelB and to Ribosomes
- Sequence of the relB transcription unit from Escherichia coli and identification of the relB gene.
- RelB is required for Peyer’s patch development: differential regulation of p52–RelB by lymphotoxin and TNF
- RelB, a new Rel family transcription activator that can interact with p50-NF-kappa B.
