Fc and C3 Receptors Induced by Herpes Simplex Virus on Cultured Human Endothelial Cells
AUTOR(ES)
Cines, Douglas B.
RESUMO
The mechanism by which immune complexes deposit in vascular tissue is uncertain. Several human viruses, including herpes simplex virus, have recently been demonstrated to replicate in human endothelial cells. Such viruses may injure vascular tissue and could play a role in the pathogenesis of immune complex deposition. Therefore, we studied the expression of receptors for immune complexes containing IgG and C3 on endothelial cells after infection with herpes simplex virus type I.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=371175Documentos Relacionados
- Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1.
- Glycoprotein gE of herpes simplex virus type 1: effects of anti-gE on virion infectivity and on virus-induced fc-binding receptors.
- Prolongation of herpes simplex virus latency in cultured human cells by temperature elevation.
- Specificity of Fc receptors induced by herpes simplex virus type 1: comparison of immunoglobulin G from different animal species.
- Herpes simplex virus type 1 persistence and latency in cultured rabbit corneal epithelial cells, keratocytes, and endothelial cells.