Glucagon release from rat pancreatic islets. A combined morphological and functional approach.
AUTOR(ES)
Carpentier, J L
RESUMO
The release of glucagon induced in isolated rat islets by arginine or by calcium deprivation has been subjected to combined functional and morphological quantifications. Arginine-stimulated glucagon release was associated with a significant increase of morphological events linked to exocytosis. By contrast, the paradoxical events linked to exocytosis. By contrast, the paradoxical release of glucagon provoked by calcium deprivation, although accompanied by a significant loss of granule stores, was not associated with an increase of morphologically detectable exocytosis.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=372471Documentos Relacionados
- Phosphoinositide metabolism and insulin secretion from isolated rat pancreatic islets.
- Possible role for calmodulin in insulin release. Studies with trifluoperazine in rat pancreatic islets.
- Rat amylin: cloning and tissue-specific expression in pancreatic islets.
- An immunocytochemical and morphometric study of the rat pancreatic islets.
- Glucose stimulation of ouabain-resistant efflux of Na+ from rat pancreatic islets.