Inactivation of the macrolide antibiotics erythromycin, midecamycin, and rokitamycin by pathogenic Nocardia species.
AUTOR(ES)
Yazawa, K
RESUMO
A survey of five Nocardia spp. with respect to susceptibility towards three macrolides (erythromycin, rokitamycin, and midecamycin) showed that the Nocardia spp. have different susceptibility profiles. Most of the resistance was due to the inactivation of the macrolides by phosphorylation, glycosylation, reduction, deacylation, or a combination thereof.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=284711Documentos Relacionados
- In vitro activity of midecamycin, a new macrolide antibiotic.
- In Vitro Development of Resistance to Erythromycin, Other Macrolide Antibiotics, and Lincomycin in Mycoplasma pneumoniae
- Effects of rokitamycin and other macrolide antibiotics on Mycoplasma pneumoniae in L cells.
- A macrolide 3-O-acyltransferase gene from the midecamycin-producing species Streptomyces mycarofaciens.
- Rate of arabinitol production by pathogenic yeast species.
