Murine cell complementation of a cold-sensitive defect for simian virus 40 replication in simian cells.
AUTOR(ES)
Fischer-Fantuzzi, L
RESUMO
Mouse 3T3 fibroblasts were found to complement, in simian cell variants semipermissive to simian virus 40, a cold-sensitive defect of an early function, but not a nonconditional defect of viral uncoating. The variant simian cells could rescue simian virus 40 from 3T3 transformants, and this capacity was not temperature dependent.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=369909Documentos Relacionados
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