Central Giant Cell Lesion
Mostrando 1-12 de 17 artigos, teses e dissertações.
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1. Immunohistochemical expression of RANKL in oral giant cell lesions is predictive of aggressiveness
Abstract The aim of this study was to evaluate the immunohistochemical expression of receptor activator of nuclear factor kappa-B ligand (RANKL) and of osteoprotegerin (OPG), important proteins correlated with osteoclastogenesis, in central giant cell lesions (CGCL) and peripheral giant cell lesions (PGCL) and to compare their expression with the histologica
Braz. oral res.. Publicado em: 29/10/2018
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2. FASN expression, angiogenesis and lymphangiogenesis in central and peripheral giant cell lesions
Central giant cell lesion (CGCL) and peripheral giant cell lesion (PGCL) are non-neoplastic proliferative processes of the jaws. PGCL is a reactive process induced by irritant local factors and CGCL is an intra-osseous lesion of unknown etiology. Both lesions exhibit similar histologic features showing abundant mononuclear cells, admixed with a large number
J. Appl. Oral Sci.. Publicado em: 2014-04
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3. Clinical and imagiological findings of central giant cell lesion and cherubism
A tomografia computadorizada de feixe cônico (TCFC) é o melhor exame para lesões ósseas da maxila, permitindo que o dentista possa avaliar com mais confiabilidade o comportamento, os componentes da lesão, e sua relação com estruturas adjacentes. A Lesão central de células gigantes e o querubismo são patologias muito semelhantes histologicamente, po
Braz. Dent. J.. Publicado em: 2013
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4. AvaliaÃÃo clÃnica da corticoterapia intralesional em lesÃo cen-tral de cÃlulas gigantes dos maxilares : relevÃncia da expressÃo dos receptores de corticÃide e calcitonina, Cox-2, p16 e amplificaÃÃo da ciclina D1 / Clinical Assessment of Intralesional Corticotherapy for Central Giant Cells Lesion Of The Jaws â The Relevance Of Steroid Receptor Expression And Calcitonin, Cox-2, P16 and Amplification of Cyclin D1. Author: Ranato Luiz Maia Nogueira. Leader: Prof. Dr. Ronaldo Albuquerque Ribeiro.
A LesÃo Central de CÃlulas Gigantes dos maxilares (LCCG) Ã intra-Ãssea, nÃo tem predileÃÃo por sexo, classifica-se em agressivas e nÃo-agressivas, histologicamente consistem tecido fi-broso e celularizado fusiforme associado a cÃlulas gigantes multinucleadas (CGM), focos de hemorragia e neovascularizaÃÃo, tendo na cirurgia seu habitual tratamento.
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 30/07/2010
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5. Exprassão imuno-histoquímica dos fatores de reabsorção óssea em lesões centrais e periféricas de células gigantes
The Giant Cell Lesions, both the Central Giant Cells Lesions (CGCL) as the Peripheral Giant Cells Lesions (PGCL), correspond to a group of oral lesions that are histologically similar entities; however they show a variable clinical behaviour. The purpose of this study was to compare the immunohistochemical expression of bone resorption factors RANK (Receptor
Publicado em: 2010
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6. Expressão imuno-histoquímica das proteínas MMP-9, VEGF e FVW em lesões centrais e periféricas de células gigantes
Lesões centrais (LCCG) e periféricas de células gigantes (LPCG) dos maxilares possuem um comportamento clínico distinto, embora compartilhem características histopatológicas semelhantes. Ainda é obscuro se essas diferenças clínicas são apoiadas por um padrão distinto de imunoexpressã o de marcadores para células gigantes multinucleadas (CG) e mo
Publicado em: 2010
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7. Mitral stenosis together with a giant cell myocarditis limited to the left atrium
A case is described in which mitral stenosis was associated with a giant cell myocarditis; the latter lesion was, however, localized to the left atrium. A quite high proportion of reported cases of giant cell myocarditis have occurred in association with rheumatic heart disease. The nature of this relationship is discussed and it is concluded that, in such c
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8. Analysis of adhesion molecules in the immunopathogenesis of giant cell arteritis.
To explore the role of adhesion molecules in mediating mononuclear cell localisation, development of the granulomatous reaction, and cell mediated damage to the arterial wall in giant cell arteritis, 17 temporal artery biopsy specimens were examined. Eleven showed the histological features of giant cell arteritis and six showed no evidence of arteritis. All
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9. Giant Lymph Node Hyperplasia of the Mediastinum (Castleman's Disease): Case Report and Review
Giant lymph node hyperplasia is a rare, benign disease involving lymph nodes in various locations, predominantly in the mediastinum. There are two variants: plasma cell (earlier and/or acute) and hyaline-vascular, more chronic with an intermediate transitional type. The usual presentation is a solitary well-circumscribed asymptomatic mass lesion, often attai
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10. Homograft replacement of aortic valve and ascending aorta in a patient with non-specific giant cell aortitis.
A case of giant cell aortitis causing ascending aortic aneurysm associated with aortic regurgitation is reported. The aneurysm was excised and the aortic valve replaced using a fresh homograft. The patient has been followed up for three and a half years. There is good evidence of correction of the haemodynamic lesion and no evidence of further arteritis or a
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11. Giant cell arteritis with spinal cord infarction and basilar artery thrombosis.
A patient with active giant cell arteritis developed paraparesis and dissociated sensory loss due to infarction in the anterior spinal artery territory at the level of T12. Three days later fatal basilar artery thrombosis occurred. No occlusive lesion was found to explain the anterior spinal artery syndrome but this was associated with active arteritis. Alte
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12. Malignancies of the Anal Margin and Perianal Skin
Malignancies of the anal margin and perianal skin are relatively uncommon lesions, comprising 3 to 4% of all anorectal malignancies. Commonly included in this group of cancers are Bowen's disease (intraepithelial squamous cell cancer), perianal Paget's disease (intraepithelial adenocarcinoma), invasive squamous cell cancer, basal cell cancer, and malignant m
Thieme Medical Publishers.