Decay Accelerating
Mostrando 1-12 de 122 artigos, teses e dissertações.
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1. RESISTÊNCIA BIOLÓGICA DA MADEIRA TRATADA DE Eucalyptus grandis e Eucalyptus cloeziana EM ENSAIOS DE LABORATÓRIO E CAMPO / BIOLOGICAL RESISTANCE OF TREATED WOOD OF Eucalyptus grandis and Eucalyptus cloeziana IN LABORATORY TESTS AND FIELD
A madeira, devido sua origem orgânica, dependendo das condições ambientais que seja exposta, pode ser deteriorada por agentes biológicos. Em vista disto se torna de extrema importância que a mesma, passe por algum tratamento preservativo, com o intuito de aumentar sua vida útil. A presente pesquisa teve por objetivo avaliar a resistência biológica da
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 19/07/2011
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2. Frequency of paroxysmal nocturnal hemoglobinuria in patients attended in Belém, Pará, Brazil
BACKGROUND: Paroxysmal nocturnal hemoglobinuria is a hematological disease with complex physiopathology. It is genetically characterized by a somatic mutation in the PIG-A gene (phosphatidylinositol glycan anchor biosynthesis, class A), in which the best known antigens are DAF (decay accelerating factor or CD55) and MIRL (membrane inhibitor of reactive lysis
Revista Brasileira de Hematologia e Hemoterapia. Publicado em: 2011-02
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3. Dark Energy and The Accelerating Universe: Conceptual Aspects and Observational Tests / Energia escura e aceleração do Universo: Aspectos conceituais e testes observacionais
In the last decade, the extraordinary progress of the astronomical observations (distances with supernovas, matter and cosmic background radiation (CBR) power spectrum, X-ray surface brightness of galaxy clusters, etc) associated with important theoretical developments turned Cosmology one of the most exciting frontiers of contemporary science. In this thesi
Publicado em: 2010
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4. rACLF, a recombinant snake venom metalloprotease, activates endothelial cells in vitro
Snake venom metalloproteases (SVMPs) comprise a family of snake venom toxins responsible for most of local and systemic effects observed during envenomation by snakes from the Viperidae family. The vascular system and more specifically the endothelium seem to be the preferential targets of these proteins. This work describes the effects of rACLF, a recombina
Journal of Venomous Animals and Toxins including Tropical Diseases. Publicado em: 2008
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5. Distribuição Temporal dos Calliphoridae (Diptera) associados à decomposição de Sus scrofa Linnaeus (Suidae) na Reserva Adolpho Ducke, Manaus, Amazonas
In this study pseudo-temporal replications were utilized to evaluate the temporal distribution of calliphoridae flies of forensic importance, associated in the decomposition processes of man-size pig models (large White, Sus scrofa L., 60 Kg), in primary Amazon rain forest in the Adolpho Ducke Forested Reserve, Manaus, Amazonas. It was identified more than t
Publicado em: 2008
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6. Trypanosoma cruzi e o Sistema Complemento: Mecanismos de ativação e o papel do gene CRIT (Complement C2 Receptor Inhibitor Trispanning) na resistência à lise em cepas de classe I e II / Trypanosoma cruzi E O SISTEMA COMPLEMENTO: MECANISMOS DE ATIVAÇÃO E O PAPEL DO GENE CRIT (COMPLEMENT C2 RECEPTOR INHIBITOR TRISPANNING) NA RESISTÊNCIA À LISE EM CEPAS DE CLASSE I E II / Trypanosoma cruzi e o Sistema Complemento: Mecanismos de ativação e o papel do gene CRIT (Complement C2 Receptor Inhibitor Trispanning) na resistência à lise em cepas de classe I e II / Trypanosoma cruzi E O SISTEMA COMPLEMENTO: MECANISMOS DE ATIVAÇÃO E O PAPEL DO GENE CRIT (COMPLEMENT C2 RECEPTOR INHIBITOR TRISPANNING) NA RESISTÊNCIA À LISE EM CEPAS DE CLASSE I E II
Trypanosoma cruzi, the agent of Chagas disease, infects 18 million people in Latin America. T. cruzi has an heteroxicenous life cycle infecting vertebrates and invertebrate hosts. Two classes of T. cruzi have been proposed based on molecular markers, the class I with a sylvatic life cycle infecting mostly marsupials, while class II parasites have a domestic
Publicado em: 2006
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7. Coxsackieviruses B1, B3, and B5 use decay accelerating factor as a receptor for cell attachment.
Receptor binding and subsequent cell-mediated internalization or disassembly are the initial steps in virus replication. Cell surface molecules that participate in this process are the primary determinants of virus tissue tropism. Monoclonal antibody blockade, immunoprecipitation, and DNA transfection were used to identify decay accelerating factor as a majo
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8. Decay-Accelerating Factor and Cytoskeleton Redistribution Pattern in HeLa Cells Infected with Recombinant Escherichia coli Strains Expressing Dr Family of Adhesins
Escherichia coli strains expressing Dr fimbriae are able to enter epithelial cells by interacting with a complement-regulatory protein, decay-accelerating factor. This model of bacterial internalization, with a well-characterized bacterial ligand and host receptor, provides a unique opportunity to investigate the early stages of invasion. We used immunofluor
American Society for Microbiology.
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9. Short Consensus Repeat Domain 1 of Decay-Accelerating Factor Is Required for Enterovirus 70 Binding
Enterovirus 70 (EV70), like several other human enteroviruses, can utilize decay-accelerating factor (DAF [CD55]) as an attachment protein. Using chimeric molecules composed of different combinations of the short consensus repeat domains (SCRs) of DAF and membrane cofactor protein (CD46), we show that sequences in SCR1 of DAF are essential for EV70 binding.
American Society for Microbiology.
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10. Solution structure of a functionally active fragment of decay-accelerating factor
The second and third modules of human decay accelerating factor (DAF) are necessary and sufficient to accelerate decay of the classical pathway (CP) convertase of complement. No structure of a mammalian protein with decay-accelerating activity has been available to date. We therefore determined the solution structure of DAF modules 2 and 3 (DAF∼2,3). Struc
National Academy of Sciences.
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11. A partial cDNA clone of trypomastigote decay-accelerating factor (T-DAF), a developmentally regulated complement inhibitor of Trypanosoma cruzi, has genetic and functional similarities to the human complement inhibitor DAF.
Resistance to complement-mediated lysis in Trypanosoma cruzi is due to the expression of complement-regulatory factors by the virulent developmental forms of this protozoan parasite. An 87- to 93-kDa molecule, which we have termed T-DAF (trypomastigote decay-accelerating factor), is present on the surface of the parasite and inhibits complement activation in
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12. Interaction with Coxsackievirus and Adenovirus Receptor, but Not with Decay-Accelerating Factor (DAF), Induces A-Particle Formation in a DAF-Binding Coxsackievirus B3 Isolate
Although many coxsackie B viruses interact with decay accelerating factor (DAF), attachment to DAF by itself is not sufficient to initiate infection. We examined the early events in infection that follow virus interaction with DAF, and with the coxsackievirus and adenovirus receptor (CAR). Interaction with soluble CAR in a cell-free system, or with CAR on th
American Society for Microbiology.