Hyperactive Behavior
Mostrando 1-12 de 18 artigos, teses e dissertações.
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1. Comportamento infantil conhecido como hiperatividade: consequência do mundo contemporâneo ou TDAH?
Child behavior known as hyperactive and usually diagnosed as ADHD (Attention deficit and hyperactivity disorder) is focused, with the purpose of verifying to what extent its origins can be related to excess of stimulation to which children are exposed in contemporary world. The hypothesis is that disorganized, disruptive and impulsive behaviors observed in m
Publicado em: 2009
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2. Efeitos da administração prolongada do esteróide anabolizante decanoato de nandrolona em comportamentos emocionais e na expressão de genes relacionados ao sistema serotoninérgico em diferentes áreas cerebrais de camundongos / Effects of prolonged administration of the anabolic-androgenic steroid nandrolone decanoate in emotional behaviors and serotonergic system related genes expression in several brain areas of mice
Nandrolone decanoate is a highly abused anabolic-androgenic steroid (AAS) by individuals looking for gains in physical strength or body appearance. Supraphysiological doses of this testosterone synthetic derivative have been associated with many physical and psychiatric adverse effects, especially reported episodes of impulsiveness and overt aggressive behav
Publicado em: 2008
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3. A concepção dos professores diante do transtorno de déficit de atenção e hiperatividade em contexto escolar: um estudo de caso.
A presente pesquisa tem como objetivo analisar a concepção do professor sobre o TDAH (Transtorno de Déficit de Atenção e Hiperatividade) na identificação, caracterização e gestão dos comportamentos desatento, hiperativo e impulsivo dos alunos em sala de aula. Os sujeitos da pesquisa foram quarenta professores que lecionam nas classes de educação
Publicado em: 2007
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4. Contribuições da análise do comportamento na avaliação e no tratamento de crianças com transtorno do défict de atenção e hiperatividade / Contributions of the Behavior Analysis to Evaluation and Treatment of Children With Attention Deficit/ Hyperactivity Disorder.
Transtorno do Déficit de Atenção e Hiperatividade (TDAH) é um fenômeno estudado em diversos países, composto de sintomas de desatenção, hiperatividade e impulsividade, ocorre na infância podendo persistir até a idade adulta. Este transtorno causa prejuízos nas áreas acadêmicas, sociais e ocupacionais, reduz a auto-estima, pode evoluir para delin
Publicado em: 2005
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5. Neurological dysfunction and hyperactive behavior associated with antiphospholipid antibodies. A mouse model.
Antiphospholipid antibodies (aPL) have been associated with various neurological manifestations, but the underlying mechanism has not been elucidated. We assessed mice with induced experimental antiphospholipid syndrome (APS) for neurological and behavioral changes. After immunization with monoclonal human anticardiolipin antibody (H-3), female BALB/c mice d
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6. Activity of the Caenorhabditis elegans UNC-86 POU transcription factor modulates olfactory sensitivity
The activity of transcription factors modulates several neural pathways that mediate complex behaviors. We describe here the role of the POU transcription factor UNC-86 in the olfactory behavior of Caenorhabditis elegans. unc-86-null mutants are defective in response to odor attractants but avoid odor repellents normally. Continuous UNC-86 activity is n
National Academy of Sciences.
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7. Insertional Mutation on Mouse Chromosome 18 with Vestibular and Craniofacial Abnormalities
A dominant mutation was generated in transgenic mice as a consequence of insertional mutation. Heterozygous mice from transgenic line 9257 (Tg(9257)) are hyperactive with bidirectional circling behavior and have a distinctive facial appearance due to hypoplasia of the nasal bone. Morphological analysis of the inner ear revealed asymmetric abnormalities of th
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8. Dibutyryl Adenosine Cyclic 3′:5′-Monophosphate Effects on Goldfish Behavior and Brain RNA Metabolism
Intraventricular administration of dibutyryl adenosine cyclic 3′:5′-monophosphate into goldfish brains produced hyperactive animals. A study of the effects of the drug (25-50 mg/kg) on the incorporation of [5-3H] orotic acid, as a precursor of labeled uridine and cytidine, into newly synthesized RNA showed the formation of an RNA with a uridine to cytidi
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9. Altered psychomotor behaviors in mice lacking pituitary adenylate cyclase-activating polypeptide (PACAP)
Pituitary adenylate cyclase-activating polypeptide (PACAP) has been conserved remarkably during evolution and is widely expressed in the mammalian brain. In Drosophila, mutation of the PACAP homologue results in behavioral defects, including impaired olfaction-associated learning and changes in ethanol sensitivity. Here, we report the generation of mice lack
The National Academy of Sciences.
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10. Expression in brain of amyloid precursor protein mutated in the alpha-secretase site causes disturbed behavior, neuronal degeneration and premature death in transgenic mice.
A double mutation in the alpha-secretase site in the betaA4 region of mouse amyloid precursor protein (APP) reduced its secretion from COS cells, polarized MDCK cells and rat primary neurons. Expression of this mutant in the brain of mice, using the neuron-specific elements of the mouse Thy-1 gene promoter, resulted in transgenic mice that became progressive
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11. Zinc inhibition of γ-aminobutyric acid transporter 4 (GAT4) reveals a link between excitatory and inhibitory neurotransmission
γ-Aminobutyric acid (GABA) transporters (GATs) play an important role in inhibitory neurotransmission by clearing synaptically released GABA and by maintaining low resting levels of GABA in synaptic and extrasynaptic regions. In certain brain regions, vesicular zinc is colocalized and coreleased with glutamate and modulates the behavior of a number of chann
National Academy of Sciences.
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12. HER2 Carboxyl-terminal Fragments Regulate Cell Migration and Cortactin Phosphorylation*
A group of breast cancer patients with a higher probability of developing metastasis expresses a series of carboxyl-terminal fragments (CTFs) of the tyrosine kinase receptor HER2. One of these fragments, 611-CTF, is a hyperactive form of HER2 that constitutively establishes homodimers maintained by disulfide bonds, making it an excellent model to study overa
American Society for Biochemistry and Molecular Biology.