Oral Dyskinesia
Mostrando 1-5 de 5 artigos, teses e dissertações.
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1. An examination of synaptic proteins following chronic haloperidol in a rat model of tardive dyskinesia
Tardive dyskinesia (TD) is a late-onset side effect mainly affecting the orofacial region of patients treated chronically with classic antipsychotic drugs such as haloperidol (HAL). The causes of TD remain unknown. We hypothesized that faulty synaptic re-organization might be related to TD-like syndromes and used the vacuous chewing movements (VCM) model in
Psychology & Neuroscience. Publicado em: 2010-12
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2. INGESTÃO DA TINTURA DE VALERIANA OFFICINALIS PROTEGE DA DISCINESIA OROFACIAL INDUZIDA POR RESERPINA EM RATOS / INTAKE OF THE VALERIANA OFFICINALIS TINCTURE PROTECTS AGAINST OROFACIAL DYSKINESIA INDUCED BY RESERPINE IN RATS
Considerando as hipóteses do papel da neurotransmissão gabaérgica e do estresse oxidativo no desenvolvimento de movimentos orais associados a neuropatologias importantes, o presente estudo investigou a possível habilidade da tintura de V. officinalis na prevenção dos movimentos de mascar no vazio (MMV) induzidos por reserpina em ratos. Os animais foram
Publicado em: 2009
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3. Effect of apomorphine on growth hormone and prolactin secretion in schizophrenic patients, with or without oral dyskinesia, withdrawn from chronic neuroleptic therapy.
Serum growth hormone (GH) and prolactin (PRL) concentrations were measured after administration of the dopamine receptor agonist, apomorphine HC1 (0.75 mg subcutaneously), to 17 chronic schizophrenic patients, four of whom had an oral dyskinesia, who were withdrawn from chronic neuroleptic therapy for periods of two to 15 weeks, and in 21 control subjects (n
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4. Oral dyskinesias associated with bilateral thalamo-capsular infarction.
Involuntary mouthing movements indistinguishable from neuroleptic-induced tardive dyskinesia followed stroke in a woman whose computed tomographic (CT) scan showed bilateral thalamo-capsular infarction.
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5. Hereditary caeruloplasmin deficiency: clinicopathological study of a patient.
A 58 year old patient with dementia, oral dyskinesia, and diabetes mellitus is described. He had an undetectable concentration of serum caeruloplasmin, as an autosomal recessive trait. Brain MRI disclosed a pronounced hypointensity in the bilateral putamina, caudate, and dentate nuclei on both T1 and T2 weighted images. Pathological findings were mainly in t