Palatogenesis
Mostrando 1-7 de 7 artigos, teses e dissertações.
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1. Expressão temporal e espacial de RECK, metaloproteinase-2 e metaloproteinase-9 durante a palatogenese em camundongos / Temporal and spatial expression of RECK, metalloproteinase-2 and metalloproteinase-9 during palatogenesis in mice
The oral clefts show a great influence in the size of their hosts. They have, since birthy, difficulties in alimentation, problems in the maxilar bones, in dental arch, in speech, and in some cases, in hearing. In addition, the hosts and their families have serious psychological problems due to the esthetic lenght caused by anomalia. With no doubt, the forma
Publicado em: 2007
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2. Cleft palate development in hamster embryos following triamcinolone treatment.
Development of the palate was studied in normal and triamcionolone-treated hamster fetuses. The results demonstrated that normal palatogenesis was completed between days 12 and 13 of gestation. Following triamcinolone treatment the reorientation of the palatal shelves was delayed before there was any general retardation of fetal growth (as indicated by crown
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3. TGF-β3-induced Palatogenesis Requires Matrix Metalloproteinases
Cleft lip and palate syndromes are among the most common congenital malformations in humans. Mammalian palatogenesis is a complex process involving highly regulated interactions between epithelial and mesenchymal cells of the palate to permit correct positioning of the palatal shelves, the remodeling of the extracellular matrix (ECM), and subsequent fus
The American Society for Cell Biology.
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4. Isolated cleft palate in mice with a targeted mutation of the LIM homeobox gene Lhx8
Formation of the mammalian secondary palate is a highly regulated and complex process whose impairment often results in cleft palate, a common birth defect in both humans and animals. Loss-of-function analysis has linked a growing number of genes to this process. Here we report that Lhx8, a recently identified LIM homeobox gene, is expressed in the mesenchym
The National Academy of Sciences.
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5. Indirect modulation of Shh signaling by Dlx5 affects the oral-nasal patterning of palate and rescues cleft palate in Msx1-null mice
Cleft palate represents one of the most common congenital birth defects in human. During embryonic development, palatal shelves display oronasal (O-N) and anteroposterior polarity before the onset of fusion, but how the O-N pattern is established and how it relates to the expansion and fusion of the palatal shelves are unknown. Here we address these question
Company of Biologists.
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6. Dissecting the genetic complexity of human 6p deletion syndromes by using a region-specific, phenotype-driven mouse screen
Monosomy of the human chromosome 6p terminal region results in a variety of congenital malformations that include brain, craniofacial, and organogenesis abnormalities. To examine the genetic basis of these phenotypes, we have carried out an unbiased functional analysis of the syntenic region of the mouse genome (proximal Mmu13). A genetic screen for recessiv
National Academy of Sciences.
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7. Palatal shelf elevation in the Wistar rat fetus.
Palatogenesis in the Wistar rat fetus was studied macroscopically, microscopically, ultrastructurally and experimentally between days 13 and 19. The developmental ages of the fetuses were calculated from the smear age of the litter adjusted for individual variations in crown-rump lengths. Palatal shelf elevation occurs at day 16.4 +/- 0.1. Experimentally ind