Parabiosis
Mostrando 1-10 de 10 artigos, teses e dissertações.
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1. Análise da origem de células precursoras de odontoblastos durante a dentinogênese reparativa / Analysis of the origin of odontoblast like cells during reparative dentinogenesis
Na polpa saudável, os odontoblastos pós-mitóticos responsáveis pela secreção de dentina primária e secundária sobrevivem durante a toda a vida do dente e são responsáveis pelas respostas às injúrias externas através da produção de dentina terciária focalmente abaixo do local da agressão. Os odontoblastos sobreviventes à injúria secretam um
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 15/12/2011
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2. Growth of Inbred Yellow (AYa) and Non-Yellow (aa) Mice in Parabiosis
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3. Prevention of type I diabetes in nonobese diabetic mice by allogenic bone marrow transplantation.
An animal model [the nonobese diabetic (NOD) mouse] for type I diabetes features a striking infiltration of T cells into the pancreatic islets. This infiltration selectively destroys beta cells. Most of the T cells are Lyt-1+, but some are Lyt-2+,3+. Transfer experiments using parabiosis revealed that insulitis can be transferred within 2 weeks after parabio
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4. Differential control of peripheral circadian rhythms by suprachiasmatic-dependent neural signals
Although dependent on the integrity of a central pacemaker in the suprachiasmatic nucleus of the hypothalamus (SCN), endogenous daily (circadian) rhythms are expressed in a wide variety of peripheral organs. The pathways by which the pacemaker controls the periphery are unclear. Here, we used parabiosis between intact and SCN-lesioned mice to show that nonne
National Academy of Sciences.
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5. Limited Multiplication of Mycobacterium lepraemurium in Parabiotic Culture, as Influenced by Osmolarity of an Alkaline-Galactomannan Medium
Kato, Laszlo (Institut de Microbiologie et d'Hygiène de l'Université de Montréal, Montreal, Quebec, Canada), and Bela Gozsy. Limited multiplication of Mycobacterium lepraemurium in parabiotic culture, as influenced by osmolarity of an alkaline-galactomannan medium. J. Bacteriol. 91:1859–1862. 1966.—Limited multiplication of Mycobacterium lepraemurium
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6. Mechanisms of immunity to respiratory syncytial virus in cotton rats.
Active immunity and maternally transmitted passive immunity to respiratory syncytial virus (RSV) were studied in cotton rats. Animals infected with respiratory syncytial virus developed complete resistance to pulmonary reinfection, which lasted at least 18 months. Nasal resistance was of shorter duration and began to diminish in 8 months. Pulmonary resistanc
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7. BMP4 regulates the hematopoietic stem cell niche
Bone morphogenetic protein 4 (BMP4) is required for mesoderm commitment to the hematopoietic lineage during early embryogenesis. However, deletion of BMP4 is early embryonically lethal and its functional role in definitive hematopoiesis is unknown. Consequently, we used a BMP4 hypomorph to investigate the role of BMP4 in regulating hematopoietic stem cell (H
American Society of Hematology.
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8. Borna disease: association with a maturation defect in the cellular immune response.
Borna disease virus (BDV) is a negative-strand RNA virus which produces persistent infection in a variety of experimental animals. In the rat, the presence or absence of clinical signs of Borna disease, a characteristic, biphasic neurobehavioral illness, depends on host-related factors. A window of opportunity exists after birth wherein inoculation with BDV
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9. Systemic alteration induced in mice by ultraviolet light irradiation and its relationship to ultraviolet carcinogenesis.
Chronic irradiation of mice with ultraviolet (UV) light produces a systemic alteration of an immunologic nature. This alteration is detectable in mice long before primary skin cancers induced by UV light begin to appear. The alteration results in the failure of UV-irradiated mice to reject highly antigenic, transplanted UV-induced tumors that are rejected by
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10. Recruitment of lymphocytes to the lung through vaccination enhances the immunity of mice exposed to irradiated schistosomes.
The effector mechanism, which operates against challenge parasites in the lungs of C57BL/6 mice vaccinated once with irradiated cercariae of Schistosoma mansoni, is mediated by CD4+ T helper lymphocytes. However, adoptive transfer of immunity from vaccinated donors to naive recipients by using sensitized T cells has not proved successful. One explanation may