Pulmonary Thromboendarterectomy
Mostrando 1-9 de 9 artigos, teses e dissertações.
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1. Pulmonary Thromboendarterectomy Without Circulatory Arrest
Abstract Introduction: Here we describe our technique and results of beating heart pulmonary thromboendarterectomy (PTE) with cardiopulmonary bypass (CPB) in four patients for treatment of chronic thromboembolic pulmonary hypertension (CTEPH). Methods: Retrospective analysis of data from patients who underwent PTE for CTEPH between January 2019 and Septemb
Brazilian Journal of Cardiovascular Surgery. Publicado em: 2022
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2. Clinical and haemodynamic evaluation of chronic thromboembolic pulmonary hypertension patients scheduled for pulmonary thromboendarterectomy: Is schistosomiasis hypertension an important confounding factor?
INTRODUCTION: Chronic thromboembolic pulmonary hypertension is a disease affecting approximately 4,000 people per year in the United States. The incidence rate in Brazil, however, is unknown. The estimated survival for patients with chronic thromboembolic pulmonary hypertension without treatment is approximately three years. Pulmonary thromboendarterectomy f
Clinics. Publicado em: 2010
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3. Pulmonary thromboendarterectomy in chronic thromboembolic pulmonary hypertension: initial experience of Hospital de Clínica de Porto Alegre
Publicado em: 2009
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4. Reatividade da artéria pulmonar, ao óxido nítrico inalado, antes e após a tromboendarterectomia / Pulmonary artery vascular reactivity, after nitric oxide inhalation, before and after pulmonary thromboendarterectomy
INTRODUCTION: After pulmonary embolism, 1 to 3% of these patients develop a chronic embolism, with elevated pulmonary artery pressure and increased pulmonary vascular resistance (PVR). The continuous elevated pressure leads to the remodeling of the small arteries. The objective of this study is to determinate if there is an increase in the responsiveness to
Publicado em: 2009
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5. Short‐term intravenous iloprost for treatment of reperfusion lung oedema after pulmonary thromboendarterectomy
This case report describes the treatment of reperfusion lung oedema after pulmonary thromboendarterectomy using intravenous iloprost infusion in a 52‐year‐old woman diagnosed with chronic thromboembolic pulmonary hypertension.
BMJ Group.
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6. Surgical treatment of pulmonary hypertension in protein C deficiency.
Two patients with protein C deficiency developed symptomatic pulmonary hypertension secondary to chronic pulmonary embolism. They were successfully treated by thromboendarterectomy.
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7. Perfusion Lung Scans Provide a Guide to Which Patients With Apparent Primary Pulmonary Hypertension Merit Angiography
There is hesitancy, based on the perceived risk, to do pulmonary angiography in patients believed to have primary pulmonary hypertension. Yet pulmonary hypertension due to major-vessel, chronic thromboembolism mimics primary pulmonary hypertension clinically and on standard laboratory tests. Because thromboembolic pulmonary hypertension is potentially remedi
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8. Inhibition of mTOR attenuates store-operated Ca2+ entry in cells from endarterectomized tissues of patients with chronic thromboembolic pulmonary hypertension
Pulmonary vascular remodeling occurs in patients with chronic thromboembolic pulmonary hypertension (CTEPH). One factor contributing to this vascular wall thickening is the proliferation of pulmonary artery smooth muscle cells (PASMC). Store-operated Ca2+ entry (SOCE) and cytosolic free Ca2+ concentration ([Ca2+]cyt) in PASMC are known to be important in cel
American Physiological Society.
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9. Identification of putative endothelial progenitor cells (CD34+CD133+Flk-1+) in endarterectomized tissue of patients with chronic thromboembolic pulmonary hypertension
Chronic thromboembolic pulmonary hypertension (CTEPH) is characterized by a fibrotic thrombus persisting and obliterating the lumen of pulmonary arteries; its pathogenesis remains poorly defined. This study investigates a potential contribution for progenitor cell types in the development of vascular obliteration and remodeling in CTEPH patients. Endarterect
American Physiological Society.