Sah Gene
Mostrando 1-10 de 10 artigos, teses e dissertações.
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1. Association of 5-HT2C (rs3813929) and UCP3 (rs1800849) gene polymorphisms with type 2 diabetes in obese women candidates for bariatric surgery
ABSTRACT Objective Obesity can cause systemic arterial hypertension (SAH) and type 2 diabetes mellitus (DM2) factor that is also influenced by genetic variability. The present study aims to investigate the association between gene polymorphisms related with obesity on the prevalence of SAH and DM2 in the preoperative period and 1 year after Roux-en-Y gastr
Arch. Endocrinol. Metab.. Publicado em: 27/03/2017
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2. Estudo da expressão do gene SAH e do gene codificador da proteína ATRAP em áreas do sistema nervoso central e sua relação com a gênese da hipertensão essencial verificada em ratos SHR / Study of SAH gene and codifying ATRAP protein gene in areas of central nervous system and your relation with essential hypertension verifying in SHR rats
A hipertensão essencial é uma doença que afeta cerca de 20% da população adulta, chegando a 50% no caso dos idosos, sendo que os mecanismos de sua gênese ainda não são totalmente conhecidos. O objetivo deste trabalho é investigar a expressão de dois genes relacionados aos mecanismos renais de controle da pressão arterial em três áreas do sistema
Publicado em: 2010
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3. The Arabidopsis HOMOLOGY-DEPENDENT GENE SILENCING1 Gene Codes for an S-Adenosyl-l-Homocysteine Hydrolase Required for DNA Methylation-Dependent Gene Silencing
Genes introduced into higher plant genomes can become silent (gene silencing) and/or cause silencing of homologous genes at unlinked sites (homology-dependent gene silencing or HDG silencing). Mutations of the HOMOLOGY-DEPENDENT GENE SILENCING1 (HOG1) locus relieve transcriptional gene silencing and methylation-dependent HDG silencing and result in genome-wi
American Society of Plant Biologists.
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4. Heme oxygenase-1 gene induction as an intrinsic regulation against delayed cerebral vasospasm in rats
Delayed cerebral vasospasm after aneurysmal subarachnoid hemorrhage (SAH) causes cerebral ischemia and infarction. To date, the pathogenesis and gene expression associated with vasospasm remain poorly understood. The present study used fluorescent differential display to identify differentially expressed genes in a rat model of SAH. By using quantitative RT-
American Society for Clinical Investigation.
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5. Involvement of S-adenosylmethionine in G1 cell-cycle regulation in Saccharomyces cerevisiae
S-adenosyl-l-methionine (AdoMet) is a molecule central to general metabolism, serving as a principal methyl donor for methylation of various cellular constituents. The alteration in the availability of AdoMet has profound effect on cell growth. A mutant allele of Saccharomyces cerevisiae gene SAH1 encoding S-adenosyl-l-homocysteine (AdoHcy) hydrolase, was is
National Academy of Sciences.
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6. pfs-Dependent Regulation of Autoinducer 2 Production in Salmonella enterica Serovar Typhimurium
Bacterial intercellular communication provides a mechanism for signal-dependent regulation of gene expression to promote coordinated population behavior. Salmonella enterica serovar Typhimurium produces a non-homoserine lactone autoinducer in exponential phase as detected by a Vibrio harveyi reporter assay for autoinducer 2 (AI-2) (M. G. Surette and B. L. Ba
American Society for Microbiology.
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7. Inactivation of CMP-N-acetylneuraminic acid hydroxylase occurred prior to brain expansion during human evolution
Humans are genetically deficient in the common mammalian sialic acid N-glycolylneuraminic acid (Neu5Gc) because of an Alu-mediated inactivating mutation of the gene encoding the enzyme CMP-N-acetylneuraminic acid (CMP-Neu5Ac) hydroxylase (CMAH). This mutation occurred after our last common ancestor with bonobos and chimpanzees, and before the origin of prese
The National Academy of Sciences.
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8. The baculovirus antiapoptotic p35 gene also functions via an oxidant-dependent pathway
Cellular imbalance in the levels of antioxidants and reactive oxygen species resulting in apoptosis is directly associated with a number of parasitic infections, aging, and several genetic and multifactorial diseases. The baculovirus AcNPV-encoded antiapoptotic p35 gene prevents apoptosis induced by a variety of apoptotic agents in different systems. We demo
The National Academy of Sciences.
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9. The mouse lethal nonagouti (a(x)) mutation deletes the S-adenosylhomocysteine hydrolase (Ahcy) gene.
The lethal nonagouti (a(x)) mutation is a hypomorphic allele of the agouti coat color locus which, when homozygous, also leads to embryonic death around the time of implantation. To understand the molecular basis of these phenotypes, we identified and cloned a deletion breakpoint junction present in the ax chromosome. Long range restriction mapping demonstra
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10. Siva-1 binds to and inhibits BCL-XL-mediated protection against UV radiation-induced apoptosis
We previously cloned Siva-1 by using the cytoplasmic tail of CD27, a member of the tumor necrosis factor receptor family, as the bait in the yeast two-hybrid system. The Siva gene is organized into four exons that code for the predominant full-length Siva-1 transcript, whereas its alternate splice form, Siva-2, lacks exon 2 coding sequence. Various groups ha
The National Academy of Sciences.