Subcortical Dementia White Matter
Mostrando 1-10 de 10 artigos, teses e dissertações.
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1. Perfil Neuropsicológico das Alterações de Linguagem nas Demências Subcorticais da Substância Branca e Cinzenta / Neuropsychological Profile of Language Changes in Dementia subcortical white matter and Grey
No século vinte acreditava-se que o prejuízo cortical era o maior substrato neuropatológico da disfunção cerebral. Os estudos de demência foram primeiramente focados nas desordens do córtex cerebral e região subcortical cinzenta, o que originou os conceitos de demência cortical e subcortical respectivamente. O relato de demência principalmente envo
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 03/08/2009
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2. Perfil Neuropsicológico das Alterações de Linguagem nas Demências Subcorticais da Substância Branca e Cinzenta / Neuropsychological Profile of Language Changes in Dementia subcortical white matter and Grey
No século vinte acreditava-se que o prejuízo cortical era o maior substrato neuropatológico da disfunção cerebral. Os estudos de demência foram primeiramente focados nas desordens do córtex cerebral e região subcortical cinzenta, o que originou os conceitos de demência cortical e subcortical respectivamente. O relato de demência principalmente envo
Publicado em: 2009
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3. Subcortical arteriosclerotic encephalopathy (Binswanger's type) and cortical infarcts in a young normotensive patient.
A 49-year-old normotensive man died after a series of strokes, slowly evolving dementia and personality change occurring over a period of 23 years. CT scan showed large infarcts involving the cortex and white matter of the temporo-occipital areas, small subcortical infarcts and low attenuation in the white matter of the frontal and parietal lobes. Neuropatho
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4. Subcortical arteriosclerotic encephalopathy: a clinical and radiological investigation.
Subcortical arteriosclerotic encephalopathy (Binswanger's type) was diagnosed in a group of patients with hypertension or arteriosclerosis, who showed acute and subacute neurological deficits, dementia, reduced cerebral blood flow, and white matter low attenuation with mild atrophy and infarcts as the predominant CT scan features. This set of clinical and ra
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5. New phenotype of the cerebral autosomal dominant arteriopathy mapped to chromosome 19: migraine as the prominent clinical feature.
A survey was carried out on a large family presenting the symptoms of familial arteriopathy (CADASIL) recently mapped to chromosome 19. This is characterised clinically by recurrent subcortical infarcts developing into pseudobulbar palsy and subcortical dementia, and radiologically by early MRI abnormalities. To characterise this familial condition, 43 membe
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6. Examination of brains of AIDS cases for human immunodeficiency virus and human cytomegalovirus nucleic acids.
The role of direct virus infection as a determining factor in acquired immunodeficiency syndrome (AIDS) dementia was investigated using in situ hybridisation for human immunodeficiency virus (HIV) and human cytomegalovirus (HCMV). Four of the five AIDS dementia patients in this series demonstrated HIV infected cells distributed in widely different parts of t
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7. High resolution SPECT with [99mTc]-d,l-HMPAO in normal pressure hydrocephalus before and after shunt operation.
Cranial CT and high resolution measurements of regional cerebral blood flow (rCBF) with brain dedicated single photon emission computer tomography (SPECT) and [99mTc]-d,l-hexamethylpropyleneamine oxime ([99mTc]-d,l-HMPAO) were performed before and after shunt operation in 14 consecutive patients with dementia and normal pressure hydrocephalus (NPH). When com
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8. Correlations of leuko-araiosis with cerebral atrophy and perfusion in elderly normal subjects and demented patients.
CT images of leuko-araiosis in brain slices were quantified according to volumes of reduced Hounsfield units in frontal periventricular white matter in groups of elderly patients with multi-infarct dementia (MID, n = 23) and dementia of the Alzheimer type (DAT, n = 16). Volumes of leuko-araiosis, estimates of atrophic cerebral tissue, and local cerebral perf
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9. Hereditary cerebral haemorrhage with amyloidosis, Dutch type (HCHWA-D): clinicopathological studies.
Clinical and neuropathological findings are reported in 63 patients with hereditary cerebral haemorrhage with amyloid angiopathy. Patients had mostly recurrent strokes, and at least 80% of these were haemorrhages. Almost a third of the patients died within a year of their first and only recorded haemorrhage, half of them within two weeks. This angiopathy was
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10. Distinct phenotypic and functional features of CADASIL mutations in the Notch3 ligand binding domain
Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) is an autosomal dominant small-vessel disease of the brain caused by mutations in the NOTCH3 receptor. The highly stereotyped nature of the mutations, which alter the number of cysteine residues within the epidermal growth factor-like repeats (EGFR), predicts
Oxford University Press.