Vana Genotype
Mostrando 1-12 de 18 artigos, teses e dissertações.
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1. CYP3A5 genotyping for assessing the efficacy of treatment with simvastatin and atorvastatin
In this work, we examined the impact of polymorphism in the cytochrome P450 (CYP) 3A5 gene, CYP3A5*1 (6986A > G, rs 776746), on the reduction in the lipid levels caused by simvastatin and atorvastatin. We studied 350 hyperlipidemic patients who received 10-40 mg of atorvastatin (n = 175) or simvastatin (n = 175) daily. Genotyping for CYP3A5 was done by PCR-R
Genet. Mol. Biol.. Publicado em: 2015-06
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2. Enterococcus gallinarum carrying the vanA gene cluster: first report in Brazil
In 2000, Enterococcus faecalis resistant to vancomycin was first reported at a tertiary hospital in Porto Alegre, southern Brazil. The resistance spread to other hospitals and surveillance programs were established by hospital infection committees to prevent the spread of vancomycin-resistant enterococci. In February 2002, an isolate initially identified at
Publicado em: 2010
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3. Epidemiologia e fatores de risco associados à colonização por VRE e MRSA em uma unidade de terapia intensiva de adultos
This investigation included a total of 78 VRE-colonized patients and 17 MRSA-colonized patients through study of the incidence in the period April 2009 to January 2010. We evaluated the rates of infection/colonization with these phenotypes, risk factors for colonization, antimicrobial susceptibility profile and characterization of vanA gene in enterococci st
Publicado em: 2010
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4. Caracterização fenotípica e molecular de fatores de virulência de Enterococcus faecium de origem hospitalar
Enterococci are Gram-positive cocci commonly found in the gastrointestinal tract of humans and animals as well as in soil, water and food. However, they are now among the leading causes of nosocomial infections. Vancomycin-resistant enterococci (VRE) were first identified in the late 1980s in a few European countries. The genes that encode the six types of a
Publicado em: 2009
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5. Molecular characterization of enterococci harboring genotype and phenotype incongruence related to glycopeptide resistance isolated in Brazilian hospitals
Three Enterococcus faecalis and one Enterococcus faecium strains were characterized by plasmid profile, pulsed-field gel electrophoresis (PFGE) and determination of antimicrobial minimal inhibitory concentrations. VanA elements were characterized by Long PCR, overlapping PCR and DNA sequencing. Enterococcal strains showed resistance to vancomycin and harbore
Memórias do Instituto Oswaldo Cruz. Publicado em: 2008-05
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6. Vancomycin-resistant van a phenotype Enterococcus faecalis: first case in Minas Gerais state and epidemiological considerations
We report the first case of vancomycin-resistant Enterococcus faecalis with VanA phenotype and vanA genotype isolated from a surgical patient in a general Hospital in Uberlândia, state of Minas Gerais, Brazil.
Brazilian Journal of Infectious Diseases. Publicado em: 2007-08
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7. Molecular characterization of the VanA element in enterococci with incongruent genotype and phenotypes relative to glycopeptide resistance. / Caracterização molecular de elementos VanA em enterococos com genótico e fenótipo discrepantes relativos à resistência aos glicopeptídeos
Enterococos resistentes aos glicopeptídeos representam, atualmente, importantes patógenos causadores de infecção nosocomial, sendo isolados em várias regiões do mundo, inclusive no Brasil. Dos fenótipos de resistência descritos até o momento, VanA e VanB são os mais encontrados. O fenótipo VanA é caracterizado por linhagens resistentes a altos n�
Publicado em: 2007
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8. Identification and molecular characterization of Van A-type vancomycin-resistant Enterococcus faecalis in Northeast of Brazil
The isolation of vancomycin resistant enterococci (VRE) in Brazil has rapidly increased, following the world wide tendency. We report in the present study the first isolation of vancomycin resistant Enterococcus faecalis (VRE) in the Northeast of Brazil. The four VRE isolates were characterized for antimicrobial susceptibility, genotypic typing by macro rest
Memórias do Instituto Oswaldo Cruz. Publicado em: 2006-11
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9. Enterococcus gallinarum carrying the vanA gene cluster: first report in Brazil
In 2000, Enterococcus faecalis resistant to vancomycin was first reported at a tertiary hospital in Porto Alegre, southern Brazil. The resistance spread to other hospitals and surveillance programs were established by hospital infection committees to prevent the spread of vancomycin-resistant enterococci. In February 2002, an isolate initially identified at
Brazilian Journal of Medical and Biological Research. Publicado em: 2004-11
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10. Emergence of vanA Genotype Vancomycin-Resistant Enterococci with Low or Moderate Levels of Teicoplanin Resistance in Korea
In Korea, vancomycin-resistant enterococci have become important nosocomial pathogens since the late 1990s, and most vancomycin-resistant enterococcal isolates have been VanA phenotype-vanA genotype strains. In 2001, we experienced an outbreak of VanB phenotype-vanA genotype vancomycin-resistant enterococci at a university hospital. This is the first report
American Society for Microbiology.
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11. Strains of glycopeptide-resistant Enterococcus faecium can alter their van genotypes during an outbreak.
Two isolates of Enterococcus faecium with VanA glycopeptide resistance were isolated during a hospital outbreak of E. faecium with plasmid-mediated VanB resistance. Both were found to be identical to the VanB outbreak strain by pulsed-field gel electrophoresis. The genotype of this strain changed from vanB to vanA through an intermediate isolate that contain
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12. Emergence of Vancomycin-Resistant Enterococci in Australia: Phenotypic and Genotypic Characteristics of Isolates
Enterococci with resistance to glycopeptides have recently emerged in Australia. We developed multiplex PCR assays for vanA, vanB, vanC1, and vanC2 or vanC3 in order to examine the genetic basis for vancomycin resistance in Australian isolates of vancomycin-resistant Enterococcus faecium and E. faecalis (VRE). The predominant genotype from human clinical E.
American Society for Microbiology.